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HN1 overexpression after Nocodazole Block (post-G2) using transient

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Last updated 24 março 2025
HN1 overexpression after Nocodazole Block (post-G2) using transient
HN1 overexpression after Nocodazole Block (post-G2) using transient
Overexpression of Rac GTPase Activating Protein 1 Contributes to Proliferation of Cancer Cells by Reducing Hippo Signaling to Promote Cytokinesis - ScienceDirect
HN1 overexpression after Nocodazole Block (post-G2) using transient
Overexpression of Rac GTPase Activating Protein 1 Contributes to Proliferation of Cancer Cells by Reducing Hippo Signaling to Promote Cytokinesis - ScienceDirect
HN1 overexpression after Nocodazole Block (post-G2) using transient
PDF) Libro B. Molecular Molecular Cell Biology Lodish, Berk, Kaiser 8 Edition
HN1 overexpression after Nocodazole Block (post-G2) using transient
Induction of cell cycle arrest and cyclin B1/Cdc2 activation in MCF-7
HN1 overexpression after Nocodazole Block (post-G2) using transient
Cell Cycle Synchronization of the Murine EO771 Cell Line Using Double Thymidine Block Treatment - Goepp - 2020 - BioEssays - Wiley Online Library
HN1 overexpression after Nocodazole Block (post-G2) using transient
Biology, Free Full-Text
HN1 overexpression after Nocodazole Block (post-G2) using transient
Cell Synchronization by Double Thymidine Block. - Abstract - Europe PMC
HN1 overexpression after Nocodazole Block (post-G2) using transient
Effect of altered HN1 expression on cell proliferation and cell cycle
HN1 overexpression after Nocodazole Block (post-G2) using transient
Overexpression of Rac GTPase Activating Protein 1 Contributes to Proliferation of Cancer Cells by Reducing Hippo Signaling to Promote Cytokinesis - ScienceDirect
HN1 overexpression after Nocodazole Block (post-G2) using transient
Cell Cycle Synchronization of the Murine EO771 Cell Line Using Double Thymidine Block Treatment - Goepp - 2020 - BioEssays - Wiley Online Library
HN1 overexpression after Nocodazole Block (post-G2) using transient
Ginkgetin induces G2-phase arrest in HCT116 colon cancer cells through the modulation of b‑Myb and miRNA34a expression
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