Capecitabine, DNA/RNA Synthesis Inhibitor

Capecitabine is an oral proagent that is converted to its active metabolite, 5-FU, by thymidine phosphorylase. - Mechanism of Action & Protocol.

How Do Antibiotics Affect Nucleic Acid Synthesis?, Biomol Blog, Resources

Molecules, Free Full-Text

5‑Fluorouracil and capecitabine therapies for the treatment of colorectal cancer (Review)

Chemotherapy

5‑Fluorouracil and capecitabine therapies for the treatment of colorectal cancer (Review)

Gemcitabine (LY 188011), DNA Synthesis Inhibitor

Mechanism of action of capecitabine upon conversion into active 5-FU.

1 PRINCIPLES OF ONCOLOGIC PHARMACOTHERAPY Elshami M. Elamin, MD Medical Oncologist Central Care Cancer Center Wichita, KS - USA. - ppt download

Capecitabine, DNA/RNA Synthesis Inhibitor

Cellular responses to replication stress: Implications in cancer biology and therapy.

Cancer & Cancer Drug

Frontiers 5-Fluorouracil: A Narrative Review on the Role of Regulatory Mechanisms in Driving Resistance to This Chemotherapeutic Agent

Systematic review of treatment intensification using novel agents for chemoradiotherapy in rectal cancer - Clifford - 2018 - BJS (British Journal of Surgery) - Wiley Online Library

Capecitabine

TAS1553, a small molecule subunit interaction inhibitor of ribonucleotide reductase, exhibits antitumor activity by causing DNA replication stress